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dc.contributor.CRUESPUNIVERSIDADE ESTADUAL DE CAMPINASpt_BR
dc.contributor.authorunicampMartins Junior, Carlos Robertopt_BR
dc.contributor.authorunicampMartinez, Alberto Rolim Muropt_BR
dc.contributor.authorunicampRezende, Thiago Junqueira Ribeiro dept_BR
dc.contributor.authorunicampBranco, Lucas de Melo Teixeirapt_BR
dc.contributor.authorunicampLopes-Cendes, Íscia Teresinhapt_BR
dc.contributor.authorunicampFrança Junior, Marcondes Cavalcantept_BR
dc.typeArtigopt_BR
dc.titleSpinal cord damage in spinocerebellar ataxia type 1pt_BR
dc.contributor.authorMartins Junior, Carlos Robertopt_BR
dc.contributor.authorMartinez, Alberto Rolim Muropt_BR
dc.contributor.authorRezende, Thiago Junqueira Ribeiro dept_BR
dc.contributor.authorBranco, Lucas Melo Teixeirapt_BR
dc.contributor.authorPedroso, Jose Luizpt_BR
dc.contributor.authorBarsottini, Orlando G. P.pt_BR
dc.contributor.authorLopes-Cendes, Isciapt_BR
dc.contributor.authorFrança Junior, Marcondes C.pt_BR
dc.subjectAtaxiapt_BR
dc.subjectEstudos transversaispt_BR
dc.subjectProgressão da doençapt_BR
dc.subjectImagem por ressonância magnéticapt_BR
dc.subject.otherlanguageAtaxiapt_BR
dc.subject.otherlanguageCross-sectional studiespt_BR
dc.subject.otherlanguageDisease progressionpt_BR
dc.subject.otherlanguageMagnetic resonance imagingpt_BR
dc.description.abstractSpinocerebellar ataxia type 1 (SCA1) is an autosomal dominant disorder caused by a CAG repeat expansion, characterized by progressive cerebellar ataxia and pyramidal signs. Non-motor and extracerebellar symptoms may occur. MRI-based studies in SCA1 focusept_BR
dc.relation.ispartofThe cerebellumpt_BR
dc.relation.ispartofabbreviationCerebellumpt_BR
dc.publisher.cityNew York, NYpt_BR
dc.publisher.countryEstados Unidospt_BR
dc.publisherSpringer Naturept_BR
dc.date.issued2017pt_BR
dc.date.monthofcirculationAug.pt_BR
dc.language.isoengpt_BR
dc.description.volume16pt_BR
dc.description.issuenumber4pt_BR
dc.description.firstpage792pt_BR
dc.description.lastpage796pt_BR
dc.rightsFechadopt_BR
dc.sourceWOSpt_BR
dc.identifier.issn1473-4222pt_BR
dc.identifier.eissn1473-4230pt_BR
dc.identifier.doi10.1007/s12311-017-0854-9pt_BR
dc.identifier.urlhttps://link.springer.com/article/10.1007%2Fs12311-017-0854-9pt_BR
dc.description.sponsorshipFUNDAÇÃO DE AMPARO À PESQUISA DO ESTADO DE SÃO PAULO - FAPESPpt_BR
dc.description.sponsorship1FAPESP - FUNDAÇÃO DE AMPARO À PESQUISA DO ESTADO DE SÃO PAULOpt_BR
dc.description.sponsordocumentnumber2013/01766-7pt_BR
dc.date.available2021-01-04T20:49:28Z-
dc.date.accessioned2021-01-04T20:49:28Z-
dc.description.provenanceMade available in DSpace on 2021-01-04T20:49:28Z (GMT). No. of bitstreams: 1 000405033000005.pdf: 612406 bytes, checksum: 3007cbe516846838198cc76ed04eb1a1 (MD5) Previous issue date: 2017en
dc.identifier.urihttp://repositorio.unicamp.br/jspui/handle/REPOSIP/353653-
dc.contributor.departmentsem informaçãopt_BR
dc.contributor.departmentsem informaçãopt_BR
dc.contributor.departmentsem informaçãopt_BR
dc.contributor.departmentsem informaçãopt_BR
dc.contributor.departmentDepartamento de Genética Médicapt_BR
dc.contributor.departmentDepartamento de Neurologiapt_BR
dc.contributor.unidadeFaculdade de Ciências Médicaspt_BR
dc.identifier.source000405033000005pt_BR
dc.creator.orcid0000-0003-2993-2402pt_BR
dc.creator.orcidsem informaçãopt_BR
dc.creator.orcid0000-0001-8453-0313pt_BR
dc.creator.orcidsem informaçãopt_BR
dc.creator.orcid0000-0002-6221-6822pt_BR
dc.creator.orcid0000-0003-0898-2419pt_BR
dc.type.formArtigo originalpt_BR
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