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Please use this identifier to cite or link to this item: http://repositorio.unicamp.br/jspui/handle/REPOSIP/352285
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dc.contributor.CRUESPUNIVERSIDADE ESTADUAL DE CAMPINASpt_BR
dc.contributor.authorunicampDias da Silveira, Wanderley-
dc.typeArtigopt_BR
dc.titlePathogenic potential and genetic diversity of environmental and clinical isolates of Pseudomonas aeruginosapt_BR
dc.contributor.authorMartins, V. V.-
dc.contributor.authorPitondo-Silva, A.-
dc.contributor.authorFreitas, S. D.-
dc.contributor.authorda Silveira, W. D.-
dc.contributor.authorFalcao, J. P.-
dc.contributor.authorde Melo Manco, L.-
dc.contributor.authorStehling, E. G.-
dc.subjectPseudomonas aeruginosapt_BR
dc.subjectVirulenciapt_BR
dc.subject.otherlanguagePseudomonas aeruginosapt_BR
dc.subject.otherlanguageVirulencept_BR
dc.description.abstractThe aim of this study was to investigate the occurrence of virulence genes among clinical and environmental isolates of Pseudomonas aeruginosa and to establish their genetic relationships by Enterobacterial Repetitive Intergenic Consensus PCR (ERIC-PCR). A total of 60 P. aeruginosa isolates from environmental and clinical sources were studied. Of these, 20 bacterial isolates were from soil, 20 from water, and 20 from patients with cystic fibrosis. Analysis of ERIC-PCR demonstrated that the isolates of P. aeruginosa showed a considerable genetic variability, regardless of their habitat. Numerous virulence genes were detected in both clinical and environmental isolates, reinforcing the possible pathogenic potential of soil and water isolates. The results showed that the environmental P. aeruginosa has all the apparatus needed to cause disease in humans and animalspt_BR
dc.relation.ispartofActa pathologica, microbiologica et immunologica scandinavicapt_BR
dc.relation.ispartofabbreviationAPMISpt_BR
dc.publisher.cityHoboken, NJpt_BR
dc.publisher.countryEstados Unidospt_BR
dc.publisherJohn Wiley & Sonspt_BR
dc.date.issued2014-
dc.date.monthofcirculationFeb.pt_BR
dc.language.isoengpt_BR
dc.description.volume122pt_BR
dc.description.issuenumber2pt_BR
dc.description.lastpage92pt_BR
dc.rightsAbertopt_BR
dc.sourceWOSpt_BR
dc.identifier.issn0903-4641pt_BR
dc.identifier.eissn1600-0463pt_BR
dc.identifier.doi10.1111/apm.12112pt_BR
dc.identifier.urlhttps://onlinelibrary.wiley.com/doi/full/10.1111/apm.12112pt_BR
dc.description.sponsorshipFUNDAÇÃO DE AMPARO À PESQUISA DO ESTADO DE SÃO PAULO - FAPESPpt_BR
dc.description.sponsordocumentnumber2009/16657-3pt_BR
dc.date.available2020-11-13T13:20:59Z-
dc.date.accessioned2020-11-13T13:20:59Z-
dc.description.provenanceSubmitted by Cintia Oliveira de Moura (cintiaom@unicamp.br) on 2020-11-13T13:20:59Z No. of bitstreams: 0. Added 1 bitstream(s) on 2021-02-16T16:52:57Z : No. of bitstreams: 1 000331081100002.pdf: 309099 bytes, checksum: 17bf0123cd3ba6f0f708340862920ff6 (MD5)en
dc.description.provenanceMade available in DSpace on 2020-11-13T13:20:59Z (GMT). No. of bitstreams: 0 Previous issue date: 2014en
dc.identifier.urihttp://repositorio.unicamp.br/jspui/handle/REPOSIP/352285-
dc.contributor.departmentDepartamento de Genética, Evolução, Microbiologia e Imunologiapt_BR
dc.contributor.unidadeInstituto de Biologiapt_BR
dc.identifier.source000331081100002pt_BR
dc.creator.orcid0000-0003-3550-0745pt_BR
dc.type.formArtigo originalpt_BR
dc.identifier.articleid100pt_BR
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